Cheick Tiote gone

[airwolex]

Tragic and really sad. May he RIP

Apparently players from sub-Saharan Africa account for 69% deaths in Federation Football yet only make up 10% of the total no of players. Apparently the problem is HCM (Hypertropic Cardiomyopathy) in athletes from Sub-Saharan Africa.

At this point, FIFA needs to throw its weight behind a study to understand this issue

RIP to the young man.

Now those stats are not a good sign.
Does anyone know if it's mostly midfield players affected, and if maybe it's a genetic heart condition that affects players from that region.
Sometimes i start thinking our black african players work too hard on the field but this is just my emotional thinking not based on scientific evidence.

I doubt if they work harder than other players. Must be genetic.

[mcal]

...did his former club NCastle found something during physical to not resign him? Reminds of Kanu's stint at Inter where they found the heart thing and reneged, but good that a follow up was done and repair made.

[Abaua1]

Someone asked earlier in the thread if he was fasting. Times of India is saying he woke up early as usual to begin his fast. Doctors in the house, over to you.

[Chief Ogbunigwe]

Someone asked earlier in the thread if he was fasting. Times of India is saying he woke up early as usual to begin his fast. Doctors in the house, over to you.

I believe he died of a heart problem, not dehydration nor starvation?

Amodu also died last year while fasting

[Thunder]

May his soul Rest In Peace!

[wiseone]

RIP Cheick and condolences to his family. Kanu was very lucky that his congenital issue was discovered early in his career.

[Goalgetter]

Another terrible loss..sudden death in a young athlete is cardiac arrest (most likely a V fib) due to HOCM (Hypertrophic Obstructive Cardiomyopathy) until proven otherwise. Tiote guy was one heck of a midfield grafter. He'll be missed big time.

[hangingfire]

RIP

Sad day for the game. We take heart knowing what a warrior Tiote was. I loved his battling spirit in Newcastle and while wearing the Elephants' shirt. Adieu Cheick! RIP.

[Goalgetter]

HOCM arises from a mutation in a gene that codes for an integral protein in heart muscles. This mutation results in abnormal heart muscles (much thicker and scarred) which ultimately results in poor electrical activity in the heart - usually sudden and sometimes silent, if lucky. Oftentimes this electrical activity leads to a fatal arrhythmia such as ventricular fibrillation - essentially the heart is unable to pump enough blood to keep vital organs alive - the main culprit being the brain. If proper CPR doesn't happen within minutes the person is essentially dead.

[YUJAM]

Interestingly enough, Tiote grew up in a place called Biafra in Treichville, Abidjan

http://www.sport-ivoire.ci/football-ele ... -cardiaque

[Comrade Machel]

There must be something in African genes causing this. Remember a lot of blacks have died in UK prisons of heart related issues. I distinctly remember a top British police commander saying there was annecdotal evidence that blacks were likely to suffer heart attack related issues when put in certain conditions eg forceful restraint and that he the policeman wanted some research to help guide the police. I liked Tiote, a lot and was very saddened to hear this when my son told me

[Coach]

^^t'is not merely a matter of arrythmia, anterior movement of the mitral valve against a thickened septum obstructs left ventricular output. Essentially, the heart can't move blood out. The relationship with arrythmia could be shortened filling time during periods of faster heart rates. The left ventricle, already of reduced compliance, doesn't stretch as much, leaving the mitral valve at higher risk of appositioning against the septum.

T'is high time CAF and alike moved beyond 12 lead ECGs and started performing ambulatory monitoring, cardiac stress tests and detailed echocardiography pre/peri/post exertion. What does a heart scan at rest show and how does that translate to cardiac performance during sustained activity and ensuing acidosis? Football has began to embrace science and greater emphasis is afforded data on physical performance. Eventually, decisions will be governed almost entirely by the sciences.

[cchinukw]

Really sad. Don't know what to say really.

[Chief Ogbunigwe]

HOCM arises from a mutation in a gene that codes for an integral protein in heart muscles. This mutation results in abnormal heart muscles (much thicker and scarred) which ultimately results in poor electrical activity in the heart - usually sudden and sometimes silent, if lucky. Oftentimes this electrical activity leads to a fatal arrhythmia such as ventricular fibrillation - essentially the heart is unable to pump enough blood to keep vital organs alive - the main culprit being the brain. If proper CPR doesn't happen within minutes the person is essentially dead.

how reliable is this? Because if the genetic mutation is known, why not screen athletes to identify those at risk? Or is this currently just a hypothesis?

[joplass]

only 30 wow...RIP brother

[jette1]

football players collapsing and passing on field of play; best physical condition anyone could possibly attain should be a challenge to the medical community

[Goalgetter]

HOCM arises from a mutation in a gene that codes for an integral protein in heart muscles. This mutation results in abnormal heart muscles (much thicker and scarred) which ultimately results in poor electrical activity in the heart - usually sudden and sometimes silent, if lucky. Oftentimes this electrical activity leads to a fatal arrhythmia such as ventricular fibrillation - essentially the heart is unable to pump enough blood to keep vital organs alive - the main culprit being the brain. If proper CPR doesn't happen within minutes the person is essentially dead.

how reliable is this? Because if the genetic mutation is known, why not screen athletes to identify those at risk? Or is this currently just a hypothesis?

Excellent point..it's actually the "leading" conclusion as at least five genes are known to be associated with HOCM. It runs in some families, but the presentation is highly variable. Screening is only currently done in people that have a strong family history - 1st degree relative with known disease or sudden death. A huge chunk of the diagnosis is made at autopsy - it goes undetected quit often as may unfortunately be the case with Tiote(RIP).

[Chief Ogbunigwe]

HOCM arises from a mutation in a gene that codes for an integral protein in heart muscles. This mutation results in abnormal heart muscles (much thicker and scarred) which ultimately results in poor electrical activity in the heart - usually sudden and sometimes silent, if lucky. Oftentimes this electrical activity leads to a fatal arrhythmia such as ventricular fibrillation - essentially the heart is unable to pump enough blood to keep vital organs alive - the main culprit being the brain. If proper CPR doesn't happen within minutes the person is essentially dead.

how reliable is this? Because if the genetic mutation is known, why not screen athletes to identify those at risk? Or is this currently just a hypothesis?

Excellent point..it's actually the "leading" conclusion as at least five genes are known to be associated with HOCM. It runs in some families, but the presentation is highly variable. Screening is only currently done in people that have a strong family history - 1st degree relative with known disease or sudden death. A huge chunk of the diagnosis is made at autopsy - it goes undetected quit often as may unfortunately be the case with Tiote(RIP).

Thanks. Yes, while screening among the general population would be resource-intensive sans a strong family history, perhaps FIFA or CAF should institute screening for athletes? I don't expect that sequencing the genes and looking for the mutations is significantly more expensive than screening athletes for drugs. Plus it would be a one-time test.

[Goalgetter]

HOCM arises from a mutation in a gene that codes for an integral protein in heart muscles. This mutation results in abnormal heart muscles (much thicker and scarred) which ultimately results in poor electrical activity in the heart - usually sudden and sometimes silent, if lucky. Oftentimes this electrical activity leads to a fatal arrhythmia such as ventricular fibrillation - essentially the heart is unable to pump enough blood to keep vital organs alive - the main culprit being the brain. If proper CPR doesn't happen within minutes the person is essentially dead.

how reliable is this? Because if the genetic mutation is known, why not screen athletes to identify those at risk? Or is this currently just a hypothesis?

Excellent point..it's actually the "leading" conclusion as at least five genes are known to be associated with HOCM. It runs in some families, but the presentation is highly variable. Screening is only currently done in people that have a strong family history - 1st degree relative with known disease or sudden death. A huge chunk of the diagnosis is made at autopsy - it goes undetected quit often as may unfortunately be the case with Tiote(RIP).

Thanks. Yes, while screening among the general population would be resource-intensive sans a strong family history, perhaps FIFA or CAF should institute screening for athletes? I don't expect that sequencing the genes and looking for the mutations is significantly more expensive than screening athletes for drugs. Plus it would be a one-time test.

FIFA for instance can mandate all registered football clubs to test their players. Perhaps that would pickup athletes at high risk of sudden death and have them get an ICD (implantable cardioverter defibrillator). At this point, given the number of players that have died, FIFA should at least have some sort of plan in the pipelines.

[bamenda boy]

Someone asked earlier in the thread if he was fasting. Times of India is saying he woke up early as usual to begin his fast. Doctors in the house, over to you.

I did ask that question. It is a sensitive issue but I think it is worth discussing.
We may see where Rohr's concerns are stemming from.

[Chief Ogbunigwe]

Someone asked earlier in the thread if he was fasting. Times of India is saying he woke up early as usual to begin his fast. Doctors in the house, over to you.

I did ask that question. It is a sensitive issue but I think it is worth discussing.
We may see where Rohr's concerns are stemming from.

Read Goalgetter's posts. Ditto Coach's. No need to trivialize a serious medical problem. Foe, Okwaraji, etc were not fasting when they died on the pitch on live tv.

[Chief Ogbunigwe]

HOCM arises from a mutation in a gene that codes for an integral protein in heart muscles. This mutation results in abnormal heart muscles (much thicker and scarred) which ultimately results in poor electrical activity in the heart - usually sudden and sometimes silent, if lucky. Oftentimes this electrical activity leads to a fatal arrhythmia such as ventricular fibrillation - essentially the heart is unable to pump enough blood to keep vital organs alive - the main culprit being the brain. If proper CPR doesn't happen within minutes the person is essentially dead.

how reliable is this? Because if the genetic mutation is known, why not screen athletes to identify those at risk? Or is this currently just a hypothesis?

Excellent point..it's actually the "leading" conclusion as at least five genes are known to be associated with HOCM. It runs in some families, but the presentation is highly variable. Screening is only currently done in people that have a strong family history - 1st degree relative with known disease or sudden death. A huge chunk of the diagnosis is made at autopsy - it goes undetected quit often as may unfortunately be the case with Tiote(RIP).

Thanks. Yes, while screening among the general population would be resource-intensive sans a strong family history, perhaps FIFA or CAF should institute screening for athletes? I don't expect that sequencing the genes and looking for the mutations is significantly more expensive than screening athletes for drugs. Plus it would be a one-time test.

FIFA for instance can mandate all registered football clubs to test their players. Perhaps that would pickup athletes at high risk of sudden death and have them get an ICD (implantable cardioverter defibrillator). At this point, given the number of players that have died, FIFA should at least have some sort of plan in the pipelines.

If you are a cardiologist (clinician or researcher) as your posts suggests, this is an avenue for a compelling scientific article which FIFA may be unable to ignore.

The thing that baffles me though is that given the number of cardiologists that these athletes see, why isn't there a greater clamor for these screening tests (though I admit our breed of scientists can be quite dogmatic).

[lukemana]

Tiote honored in China

More on http://news.abidjan.net/p/444645.html

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CIV national team with Tiote's family
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